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dc.contributor.authorÖztürk, Yusuf
dc.contributor.authorAltan, Vecdi Melih
dc.contributor.authorYıldızoğlu-Arı, N.
dc.date.accessioned2019-10-19T14:43:55Z
dc.date.available2019-10-19T14:43:55Z
dc.date.issued1992
dc.identifier.issn0901-9928
dc.identifier.urihttps://dx.doi.org/10.1111/j.1600-0773.1992.tb00978.x
dc.identifier.urihttps://hdl.handle.net/11421/13295
dc.descriptionPubMed: 1360656en_US
dc.description.abstractAbstract: Recently, decreased gastrointestinal ß-adrenergic responses in experimental diabetes have been demonstrated. Gastrointestinal responses to ß-adrenoceptor agonists are impaired in both insulin-dependent and non-insulin-dependent diabetic rat. Insulin treatment improves the impaired gastrointestinal ß-adrenergic responsiveness of diabetic rats. The improvement seen with insulin treatment on ß-adrenergic responsiveness is closely related to protein biosynthesis. The decreased ß-adrenergic responses in diabetic rat gastrointestinal tract seem to result from a decrease in the number of ß-adrenoceptors. It is most likely that the decreased gastrointestinal ß-adrenergic responsiveness is related to an impairment in the turnover of ß-adrenoceptors as a consequence of diabetes and that insulin has a beneficial effect on the impaired receptor turnover. 1992 Nordic Pharmacological Societyen_US
dc.language.isoengen_US
dc.relation.isversionof10.1111/j.1600-0773.1992.tb00978.xen_US
dc.rightsinfo:eu-repo/semantics/openAccessen_US
dc.titleß-Adrenergic Responsiveness of the Gastrointestinal Tract in Diabetic Ratsen_US
dc.typereviewen_US
dc.relation.journalPharmacology & Toxicologyen_US
dc.contributor.departmentAnadolu Üniversitesi, Eczacılık Fakültesi, Farmakoloji Anabilim Dalıen_US
dc.identifier.volume71en_US
dc.identifier.issue4en_US
dc.identifier.startpage250en_US
dc.identifier.endpage253en_US
dc.relation.publicationcategoryDiğeren_US
dc.contributor.institutionauthorÖztürk, Yusuf


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